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In research, it’s extremely difficult to do any long term, diet-controlled studies due to lack of compliance. Irrespective of the diet, you can imagine controlling subjects for several years is a difficult task for any researcher to do. That being said, there are several long-term studies showing a positive benefit for the ketogenic diet. One study found that 66 obese individuals with high cholesterol were able to lower their weight and cholesterol levels with a ketogenic diet over 56 weeks. [4] A meta analysis (which is an analysis of several published papers) found that “Individuals assigned to a ketogenic diet achieve a greater weight loss than those assigned to a low fat diet in the long term; hence, a ketogenic diet may be an alternative tool against obesity. ” [5] All of the studies analyzed were greater than 12 months in duration. Lastly, even reducing your carbohydrate intake to less than 20% has shown to have sustainable weight loss even 44 months later! [6]For individuals starting a keto diet in the 1990’s, adherence to the diet was likely a lot harder as options were a bit more limited. Today, you can “ketofy” nearly anything from pizza to desserts so it’s much more sustainable and the research shows the weight loss is maintained. Nutritional Completeness: 1. 6/5 Stars Rated as “extremely incomplete” in the nutrition category, panelists had concerns about the saturated fat content. One expert explained that ‘any diet that recommends snacking on bacon can’t be taken seriously as a health-promoting way to eat’ and another added that the keto diet is ‘nutritionally unbalanced and likely difficult for most people to stick with long-term, which means that any weight loss will likely be regained.
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[4]GLUT 1 Insulin independentBloodBlood-brain-barrierHeart (partially) GLUT 2Insulin-independentLiverPancreasSmall IntestineGLUT 3Insulin-independentBrain NeuronsSpermGLUT 4Insulin-dependentSkeletal MuscleAdipose tissue (fat)HeartDysfunctions in Glucose RegulationGlucose regulation can become dysfunctional at many different steps. If insulin is not produced sufficiently, glucose is unable to enter insulin-dependent cells and these cells can starve. This is known as type 1 diabetes mellitus. If insulin is produced sufficiently, but receptors are damaged or are insulin resistant, they can not signal to allow glucose transport, and again cells become starved for energy. This is known as type 2 diabetes. [5]What is Gestational Diabetes?Gestational diabetes is dysfunction in blood glucose regulation specifically in pregnant women.
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[2] Contrary to
popular belief, not all cells require insulin in order to transport glucose inside of a cell. Some organs and tissues are insulin-independent, meaning insulin is not required, whereas others are insulin-dependent, meaning they require insulin. [3] GLUT 4 transporters are located on skeletal muscle,
adipose tissue, and the heart and do require insulin. In order for glucose to be transported into these cells, insulin must bind to insulin receptors and signal for this process to occur. Insulin is produced by beta cells in the pancreas in response to high blood glucose levels. Once it is released, it binds to insulin receptors and triggers a response to allow glucose to be transported into the cell. [4]GLUT 1 Insulin independentBloodBlood-brain-barrierHeart (partially) GLUT 2Insulin-independentLiverPancreasSmall IntestineGLUT 3Insulin-independentBrain NeuronsSpermGLUT 4Insulin-dependentSkeletal MuscleAdipose tissue (fat)HeartDysfunctions in Glucose RegulationGlucose regulation can become dysfunctional at many different steps. If insulin is not produced sufficiently, glucose is unable to enter insulin-dependent cells and these cells can starve. This is known as type 1 diabetes mellitus. If insulin is produced sufficiently, but receptors are damaged or are insulin resistant, they can not signal to allow glucose transport, and again cells become starved for energy. This is known as type 2 diabetes.
