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Within just six weeks of being on a ketogenic diet, researchers found that the “ketogenic diet does not have a deleterious effect on cardiovascular disease risk profile and may improve the lipid disorders characteristic of atherogenic dyslipidemia. ”  Not only may it not have negative effects on your heart, but it might significantly help. As mentioned in this review article, “At low concentration, endogenously produced ketone bodies upon uptake of a ketogenic diet or supplemented ketone bodies (or their precursors) may prove beneficial to ameliorate endothelial function and, consequently, pathologies in which endothelial damage occurs. ” We are going to have an MD speak on this exact topic at our upcoming Global Virtual Keto Summit. Make sure you don’t miss it!Managing or Preventing Diabetes 2. 6/5 StarsThe panel of experts stated that preventing or controlling diabetes with the keto diet was minimally effective.
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One expert went as far to say that the ketogenic diet isn’t safe for those with diabetes. Ironically, the explanation in the article did include the disclaimer that research has proven that decreasing carbohydrates can help with the disease. Counter: Of all the areas for these experts to lend support to a low carb, ketogenic diet, this should be the one. The data is OVERWHELMINGLY clear that a ketogenic diet can not only help prevent diabetes but actually has been shown to reverse it. Despite the fact that the CDC still recommends that those with type II diabetes consume 225 grams of carbohydrates per day, doctors are speaking out against this recommendation as it keeps patients on the vicious cycle of diabetes medication requiring carbohydrates to prevent hypoglycemia, followed by needing the medication for insulin that is released after the consumed carbs. A recent meta-analysis which included 20 years of published research studying the effects of a ketogenic diet on patients with type 2 diabetes. The review concluded that ketogenic diet is ‘superior’ in terms of glycemic control—the results were significant enough to recommend the keto diet as a treatment for type 2 diabetes. Another recent study concludes that in addition to reduction in body weight and improving lipid profile, there was a significant improvement in HbA1c and reduction in the intake of insulin and oral antidiabetic drugs in patients with type 2 diabetes.  Not only does this research show how the keto diet can lead to weight loss and improved metabolic health, it’s also monetarily cost effective as it reduces the need for insulin and oral antidiabetic drugs. Lastly, our friends at Virta Health are doing incredible work reversing type 2 diabetes with their low carb protocols. One of their studies shows that this dietary monitoring protocol lowered glycemic control medication from 55.
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While glucose is a simple sugar, it is still a relatively large molecule, therefore it needs certain transporters
to allow it to enter a cell. These are known as GLUTs (glucose transporters).  Contrary to popular belief, not all cells require
insulin in order to transport glucose inside of a cell. Some organs and tissues are insulin-independent, meaning insulin is not required, whereas others are insulin-dependent, meaning they require insulin.  GLUT 4 transporters are located on skeletal muscle, adipose tissue, and the heart and do require insulin. In order for glucose to be transported into these cells, insulin must bind to insulin receptors and signal for this process to occur. Insulin is produced by beta cells in the pancreas in response to high blood glucose levels. Once it is released, it binds to insulin receptors and triggers a response to allow glucose to be transported into the cell. GLUT 1 Insulin independentBloodBlood-brain-barrierHeart (partially) GLUT 2Insulin-independentLiverPancreasSmall IntestineGLUT 3Insulin-independentBrain NeuronsSpermGLUT 4Insulin-dependentSkeletal MuscleAdipose tissue (fat)HeartDysfunctions in Glucose RegulationGlucose regulation can become dysfunctional at many different steps. If insulin is not produced sufficiently, glucose is unable to enter insulin-dependent cells and these cells can starve. This is known as type 1 diabetes mellitus.